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Gpbar1 agonism promotes a Pgc-1-dependent browning of white adipose tissue and energy expenditure and reverses diet-induced steatohepatitis in mice

The HS-enhanced salt tolerance regulation model developed in this study requires the core pathways and candidate genes to be functionally validated through in vivo experiments, such as gene editing

Hispanic: 828.32, 925.76, 1109.98, 1322.31 (0.98, 0.01)

Using a concentration range of 0.0022 nM, 13 nm cAuNPs induced apoptosis in rabbit articular chondrocytes and no effects were observed for 3 and 45 nm cAuNPs under the same conditions

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Notably, NINJ1 deficiency did not protect against cell death (as measured by ATP) induced by RSL3 and ML162 [33,34,35], consistent with our observation that NINJ1 knockdown had no significant impact on RSL3-induced cell death